Pneumothorax Clinical Signs and TMC CSE Exam Tips Vector

Pneumothorax Signs, Symptoms, and Clinical Clues

by | Updated: Jul 15, 2026

A pneumothorax occurs when air enters the pleural space, which is the thin space between the lung and the chest wall. This air disrupts the normal negative pressure that keeps the lung expanded, allowing part or all of the affected lung to collapse.

The signs of a pneumothorax can range from mild discomfort to severe respiratory distress, depending on its size, cause, speed of development, and the patient’s underlying lung function.

Recognizing the signs early is essential because a tension pneumothorax can quickly become life-threatening.

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What is a Pneumothorax?

A pneumothorax is the presence of air in the pleural space. Under normal conditions, the pleural space contains only a small amount of fluid and maintains negative pressure that helps keep the lungs expanded during breathing. When air enters this space, the negative pressure is lost, and the lung may partially or completely collapse.

This collapse reduces ventilation on the affected side and may impair oxygenation. In a small pneumothorax, the patient may have mild symptoms or even no symptoms at all. In a larger pneumothorax, the patient may develop chest pain, shortness of breath, decreased breath sounds, and signs of respiratory distress.

A pneumothorax may occur spontaneously, after trauma, during mechanical ventilation, or as a complication of invasive procedures. It can also occur in patients with underlying lung disease, such as emphysema, COPD, asthma, cystic fibrosis, or lung infections. These patients often tolerate even a small pneumothorax poorly because their pulmonary reserve is already reduced.

The most dangerous form is a tension pneumothorax. This occurs when air enters the pleural space and becomes trapped under pressure. As pressure builds, the affected lung collapses further, the mediastinum shifts, venous return to the heart decreases, cardiac output falls, and the patient may develop shock. A tension pneumothorax is a medical emergency that requires immediate treatment.

Pneumothorax Clinical Signs Illustration Infographic

Why the Signs of a Pneumothorax Matter

The signs of a pneumothorax are important because the condition can worsen quickly, especially in trauma patients or those receiving positive-pressure ventilation. A simple pneumothorax may impair breathing, but a tension pneumothorax can rapidly compromise both ventilation and circulation.

In respiratory care, pneumothorax is often identified through a combination of symptoms, physical assessment findings, ventilator changes, oxygenation changes, and imaging. No single sign tells the entire story in every patient. Instead, clinicians look for patterns.

For example, sudden chest pain, shortness of breath, decreased breath sounds on one side, and hyperresonance to percussion strongly suggest pneumothorax. If these findings are accompanied by hypotension, tachycardia, tracheal deviation, and worsening hypoxemia, tension pneumothorax should be suspected.

This distinction matters because stable patients may undergo chest imaging to confirm the diagnosis, while unstable patients with suspected tension pneumothorax require immediate decompression. Waiting for imaging in a deteriorating patient can delay lifesaving treatment.

Sudden Chest Pain

Sudden chest pain is one of the most common signs of a pneumothorax. The pain is often sharp, stabbing, or pleuritic, meaning it worsens with deep breathing, coughing, or movement. It is usually felt on the same side as the pneumothorax.

This pain occurs because air in the pleural space irritates the pleural membranes and disrupts normal lung expansion. The affected lung cannot expand properly, and the chest wall may move differently on that side.

In a spontaneous pneumothorax, the chest pain may occur suddenly in an otherwise stable person. In a trauma patient, the pain may occur after blunt or penetrating chest injury. In a mechanically ventilated patient, chest pain may not be reported if the patient is sedated or unable to communicate, so other clinical signs become more important.

Chest pain alone does not confirm pneumothorax because many conditions can cause similar symptoms. Pulmonary embolism, myocardial infarction, pleurisy, pneumonia, rib fracture, and musculoskeletal injury may also cause chest pain. However, when sudden chest pain occurs with dyspnea and unilateral changes in breath sounds, pneumothorax should be considered.

Shortness of Breath

Dyspnea, or shortness of breath, is another major sign of pneumothorax. The patient may feel unable to take a full breath, may breathe faster than normal, or may appear visibly distressed.

The severity of dyspnea depends on several factors, including the size of the pneumothorax, the speed at which it develops, and the patient’s baseline lung function. A small pneumothorax in a healthy person may cause only mild shortness of breath. In contrast, a small pneumothorax in a patient with COPD, pulmonary fibrosis, or another chronic lung disease may cause severe respiratory distress.

This happens because patients with underlying lung disease have less reserve. They may already rely heavily on limited functional lung tissue. When part of one lung collapses, their ability to compensate is reduced.

Shortness of breath may also worsen if the pneumothorax enlarges. For this reason, increasing dyspnea during observation is an important warning sign. A patient who initially appears stable but later reports worsening breathlessness should be reassessed immediately.

Increased Respiratory Rate

Tachypnea, or an increased respiratory rate, commonly occurs when a pneumothorax interferes with ventilation and oxygenation. The body responds by increasing the breathing rate in an attempt to maintain gas exchange.

In mild cases, tachypnea may be subtle. In more severe cases, the patient may appear anxious, restless, or unable to speak in full sentences. The use of accessory muscles may also be present, especially when the patient is struggling to move air.

Tachypnea is especially concerning when it occurs along with falling oxygen saturation, unilateral absent breath sounds, chest asymmetry, or signs of poor perfusion. These findings may indicate that the pneumothorax is expanding or progressing toward tension physiology.

Note: An increased respiratory rate combined with sudden chest pain, unilateral hyperresonance, and diminished breath sounds is a classic pattern for pneumothorax.

Diminished or Absent Breath Sounds

Diminished or absent breath sounds on the affected side are among the most important bedside signs of a pneumothorax. Because air in the pleural space prevents the lung from expanding normally, ventilation to that area is reduced or absent.

During auscultation, the clinician may hear decreased breath sounds over the affected lung field. In a large pneumothorax, breath sounds may be absent. This finding is especially important because it can be detected quickly at the bedside.

Unilateral decreased breath sounds should always prompt further assessment. Pneumothorax is one possible cause, but other conditions can produce similar findings. These include right mainstem intubation, mucus plugging, pleural effusion, atelectasis, or unilateral lung disease.

Note: In a mechanically ventilated patient, sudden unilateral absent breath sounds are particularly concerning. If the patient also has high airway pressures, worsening oxygen saturation, or hypotension, tension pneumothorax must be considered.

Hyperresonance to Percussion

Hyperresonance to percussion is a classic physical sign of pneumothorax. Percussion involves tapping on the chest wall to assess the underlying air content of the thorax.

Normal lung tissue produces a resonant sound because it contains air. In pneumothorax, excess air is present in the pleural space, which can produce a louder, more hollow, drum-like sound called hyperresonance.

This finding helps distinguish pneumothorax from other conditions. For example, pleural effusion, pneumonia, and atelectasis often produce dullness to percussion because fluid, tissue density, or collapse replaces normal air-filled lung. Pneumothorax, on the other hand, produces increased resonance due to trapped pleural air.

Note: Unilateral hyperresonance is especially suspicious. Bilateral hyperresonance may occur in obstructive lung diseases such as asthma or emphysema, but hyperresonance on only one side supports the possibility of pneumothorax.

Asymmetrical Chest Movement

Asymmetrical chest movement is another important sign. Normally, both sides of the chest expand evenly during inspiration. When a pneumothorax is present, the affected side may move less because the lung is partially or completely collapsed.

This reduced movement may be seen during inspection or felt during palpation. The clinician may observe that one side of the chest rises less than the other. In larger pneumothoraces, the asymmetry may be obvious.

Asymmetrical chest movement is not specific to pneumothorax. It may also occur with flail chest, pleural effusion, atelectasis, unilateral diaphragmatic dysfunction, or right mainstem intubation. However, when it occurs with sudden dyspnea, chest pain, decreased breath sounds, and hyperresonance, pneumothorax becomes a leading concern.

In trauma patients, asymmetrical chest movement should be taken seriously because chest wall injuries and pneumothorax may occur together. A patient with blunt or penetrating chest trauma can develop a pneumothorax that worsens over time, especially if positive-pressure ventilation is required.

Decreased Tactile Fremitus

Tactile fremitus refers to vibrations felt on the chest wall when the patient speaks. Under normal conditions, sound vibrations travel through the airways and lung tissue to the chest wall. In pneumothorax, air in the pleural space separates the lung from the chest wall, which reduces the transmission of vibrations.

As a result, tactile fremitus may be decreased over the affected area. This finding can support the diagnosis when combined with other assessment data.

Decreased tactile fremitus may also occur with pleural effusion, airway obstruction, or severe hyperinflation. Therefore, it should not be interpreted alone. However, when decreased tactile fremitus appears with hyperresonance, decreased breath sounds, and sudden dyspnea, pneumothorax should be suspected.

This is a useful exam point because different lung conditions affect fremitus in different ways. Consolidation, such as pneumonia, may increase tactile fremitus because dense lung tissue transmits vibrations better. Pneumothorax usually decreases fremitus because pleural air blocks transmission.

Falling Oxygen Saturation

A decrease in oxygen saturation can be an early warning sign that a pneumothorax is worsening. As the affected lung collapses, ventilation to that region decreases. Blood may continue to pass through poorly ventilated lung tissue, contributing to ventilation-perfusion mismatch and shunting.

In a small pneumothorax, oxygen saturation may remain normal, especially in a healthy patient. However, worsening hypoxemia is concerning. It may indicate that the pneumothorax is enlarging, the patient is tiring, or tension physiology is developing.

Monitoring oxygen saturation is especially important during observation. If a patient with a known pneumothorax becomes more short of breath or their oxygen saturation falls, the condition may no longer be stable. This may require drainage of the pleural air.

In mechanically ventilated patients, falling oxygen saturation may occur suddenly. When this happens with high-pressure alarms, unilateral diminished breath sounds, or hypotension, pneumothorax should be included in the differential diagnosis.

Tachycardia

Tachycardia, or an increased heart rate, is a common sign of respiratory distress and physiologic stress. In pneumothorax, tachycardia may occur because the patient is hypoxemic, anxious, in pain, or developing impaired venous return.

In a simple pneumothorax, the heart rate may be mildly elevated. In tension pneumothorax, tachycardia can become more pronounced as intrathoracic pressure rises and circulation becomes compromised.

The presence of tachycardia should be interpreted with the full clinical picture. A patient with sudden dyspnea, chest pain, decreased breath sounds, hyperresonance, and tachycardia may have a pneumothorax. If hypotension is also present, tension pneumothorax should be strongly suspected.

Note: Tachycardia is one of the key signs that helps separate a stable pneumothorax from a more severe presentation. It may be one of the earliest signs that the patient is deteriorating.

Hypotension

Hypotension is a serious sign, especially when tension pneumothorax is possible. In tension pneumothorax, air becomes trapped in the pleural space under pressure. As pressure rises, it pushes mediastinal structures toward the opposite side and compresses the great vessels.

This reduces venous return to the right side of the heart. When less blood returns to the heart, cardiac output falls. As cardiac output decreases, blood pressure drops.

Hypotension in this setting is a sign of obstructive shock. It indicates that the pneumothorax is no longer just a respiratory problem. It has become a circulatory emergency.

A patient with hypotension, tachycardia, respiratory distress, absent breath sounds on one side, and hyperresonance should be treated immediately if tension pneumothorax is suspected. Imaging should not delay treatment in an unstable patient.

Tracheal Deviation

Tracheal deviation is a classic sign of tension pneumothorax, although it is often a late finding. In a tension pneumothorax, increasing pressure in the pleural space pushes the mediastinum away from the affected side. This may cause the trachea to shift toward the unaffected side.

For example, a right-sided tension pneumothorax may push the trachea to the left. A left-sided tension pneumothorax may push the trachea to the right.

Although tracheal deviation is often emphasized in textbooks and board exam questions, clinicians should not wait for this sign before taking action. It may be difficult to detect, especially in obese patients, trauma patients, or those with neck swelling. It may also occur late, after significant pressure has already developed.

Note: Tracheal deviation is most concerning when it appears with hypotension, tachycardia, severe dyspnea, unilateral absent breath sounds, and hyperresonance. This pattern strongly suggests tension pneumothorax.

Jugular Venous Distention

Jugular venous distention (JVD) may occur in tension pneumothorax when increased intrathoracic pressure impairs venous return to the heart. Blood backs up into the venous system, causing the neck veins to appear distended.

JVD is not always present. It may be difficult to see in patients with obesity, neck trauma, low blood volume, or poor lighting. In some cases, hypotension may occur without obvious jugular venous distention.

When present, JVD can support the diagnosis of tension pneumothorax, especially when combined with respiratory distress and unilateral chest findings. However, JVD can also occur with cardiac tamponade, right-sided heart failure, pulmonary embolism, and other conditions.

Note: For exam purposes, JVD plus hypotension plus absent breath sounds on one side should raise concern for tension pneumothorax, especially after trauma or during mechanical ventilation.

Cyanosis

Cyanosis is a bluish discoloration of the skin, lips, or nail beds caused by severe hypoxemia. It is a late and serious sign of inadequate oxygenation.

In pneumothorax, cyanosis may occur when the affected lung is significantly collapsed or when tension physiology compromises ventilation and circulation. It indicates that oxygen delivery to the tissues is impaired.

Cyanosis should never be considered an early screening sign. A patient may be dangerously hypoxemic before cyanosis becomes obvious. Pulse oximetry and clinical assessment are more reliable for early detection.

Note: When cyanosis appears with severe respiratory distress, falling oxygen saturation, tachycardia, hypotension, and unilateral absent breath sounds, emergency intervention may be required.

Subcutaneous Emphysema

Subcutaneous emphysema occurs when air tracks into the soft tissues under the skin. It may be felt as a crackling sensation during palpation, often described as crepitus. This sensation may be present around the chest wall, neck, face, or upper torso.

Subcutaneous emphysema can occur with pneumothorax, pneumomediastinum, chest trauma, or air leaks after procedures. It may also occur after chest tube placement or positive-pressure ventilation.

The presence of subcutaneous air does not always prove that a pneumothorax is present. However, it indicates that air has escaped into abnormal tissue planes, which means the patient may be at risk for pneumothorax or another air leak syndrome.

In a trauma patient, crepitus over the chest wall should prompt careful assessment for pneumothorax. If the patient also has dyspnea, unilateral decreased breath sounds, and hyperresonance, the suspicion becomes stronger.

Signs in Mechanically Ventilated Patients

Pneumothorax can be especially dangerous in patients receiving mechanical ventilation. Positive-pressure ventilation can force more air into the pleural space if an alveolar or bronchial tear is present. This can convert a simple pneumothorax into a tension pneumothorax.

One major sign is a sudden increase in peak inspiratory pressure. This may occur because the lung becomes less compliant as air accumulates in the pleural space and compresses the lung.

Plateau pressure may also rise if lung compliance decreases. In volume-control ventilation, both peak and plateau pressures may increase when a pneumothorax causes reduced compliance. In pressure-control ventilation, the delivered tidal volume may suddenly fall because the ventilator is delivering a set pressure, but the lung is less able to accept volume.

Other signs may include worsening oxygen saturation, decreased tidal volume, high-pressure alarms, sudden hypotension, unilateral absent breath sounds, and chest asymmetry.

However, increased peak pressure is not specific to pneumothorax. It may also result from airway secretions, bronchospasm, biting the endotracheal tube, patient-ventilator asynchrony, airway obstruction, or right mainstem intubation. This is why rapid bedside assessment is necessary.

Signs of Tension Pneumothorax

Tension pneumothorax is a life-threatening emergency. It occurs when air enters the pleural space and cannot escape. The trapped air builds pressure with each breath, causing progressive lung collapse and mediastinal shift.

The key signs include sudden respiratory distress, absent or diminished breath sounds on the affected side, hyperresonance to percussion, tachycardia, hypotension, tracheal deviation away from the affected side, and worsening hypoxemia.

The patient may also have jugular venous distention, cyanosis, anxiety, restlessness, or signs of shock. If untreated, tension pneumothorax can progress to cardiovascular collapse, pulseless electrical activity, and death.

A major clinical point is that tension pneumothorax is often diagnosed at the bedside. If the patient is unstable and the signs strongly suggest tension pneumothorax, treatment should not be delayed for a chest x-ray or CT scan. Immediate needle decompression or chest tube placement is required.

Chest X-Ray Signs

A chest x-ray is commonly used to confirm pneumothorax in stable patients. On an upright chest radiograph, a pneumothorax may appear as a visible pleural line near the outer edge of the lung. Beyond that line, there are no lung markings because air fills the pleural space.

The affected lung may appear partially collapsed toward the hilum. The air-filled pleural space appears darker than normal because air allows more x-rays to pass through.

In tension pneumothorax, the chest x-ray may show mediastinal shift away from the affected side, depression of the hemidiaphragm, and compression of the opposite lung.

In supine patients, pneumothorax can be harder to detect. One possible sign is the deep sulcus sign, which appears as an abnormally deep and dark costophrenic angle. This is important in trauma and ICU patients who may not be able to sit upright for imaging.

Note: A chest x-ray is useful, but it should not delay emergency treatment when tension pneumothorax is strongly suspected in an unstable patient.

Ultrasound Signs

Ultrasound is often used in emergency and critical care settings to help detect pneumothorax quickly. It is especially useful when a patient is unstable or when transporting the patient to radiology would be difficult.

One important ultrasound sign is the absence of lung sliding. Normally, the visceral and parietal pleura slide against each other during breathing. In pneumothorax, air separates the pleural layers, so lung sliding may disappear.

Another finding is the absence of B-lines. B-lines are vertical artifacts that arise from the pleural line and move with lung sliding. Because pneumothorax separates the pleural surfaces, B-lines are typically absent in the affected area.

The lung point is another important ultrasound finding. It represents the transition between normal pleural sliding and the area of pneumothorax. When identified, it is highly specific for pneumothorax.

Ultrasound findings should always be interpreted with the patient’s clinical condition. Absence of lung sliding can occur for reasons other than pneumothorax, such as mainstem intubation, pleural adhesions, apnea, or severe lung disease.

CT Scan Findings

A CT scan is the most sensitive imaging test for pneumothorax. It can detect small pneumothoraces that may not appear on a standard chest x-ray. CT is especially helpful in trauma patients, postoperative patients, or patients with complex lung disease.

CT imaging may show the exact location and size of the pneumothorax. It can also help distinguish pneumothorax from large bullae, blebs, pneumomediastinum, or other abnormalities.

This distinction is important in patients with emphysema because large bullae can sometimes resemble pneumothorax on chest x-ray. CT can clarify the diagnosis when the x-ray is uncertain.

Although CT is highly sensitive, it is not always the first choice in an unstable patient. If tension pneumothorax is suspected and the patient is deteriorating, treatment comes first.

Signs in Neonates

Pneumothorax can be more difficult to recognize in neonates because breath sounds transmit widely through the small chest. This makes it harder to identify unilateral decreased breath sounds by auscultation alone.

Signs may include sudden respiratory distress, increased oxygen requirement, cyanosis, tachypnea, grunting, retractions, or sudden deterioration during ventilatory support. Heart sounds may shift away from the side of the pneumothorax, which can provide an important clue.

Some centers may use transillumination to help detect pneumothorax in neonates. This involves shining a high-intensity light through the chest wall. Excess air in the pleural space may allow abnormal light transmission. However, imaging and clinical assessment remain important for confirmation and management.

Note: Neonates receiving positive-pressure ventilation are at increased risk because fragile lung tissue can be injured by pressure or volume. Sudden deterioration in a ventilated newborn should prompt assessment for pneumothorax.

Differentiating Pneumothorax from Similar Conditions

Several conditions can mimic the signs of pneumothorax. This is why clinicians must evaluate the entire pattern of findings.

Pleural effusion can cause decreased breath sounds and reduced chest movement, but percussion is usually dull rather than hyperresonant. Atelectasis can also cause decreased breath sounds, but the trachea may shift toward the affected side if there is significant volume loss. In tension pneumothorax, the trachea typically shifts away from the affected side.

Right mainstem intubation can cause absent breath sounds on the left side and worsening oxygenation. This is especially important in intubated patients. The endotracheal tube position should be checked when unilateral breath sounds are present.

Bronchospasm can cause increased peak airway pressure, wheezing, and difficulty ventilating, but breath sounds are usually present bilaterally. Mucus plugging may cause unilateral decreased breath sounds and atelectasis, but percussion findings and imaging can help distinguish it from pneumothorax.

Note: The key is to recognize the cluster of signs: sudden dyspnea, chest pain, unilateral decreased or absent breath sounds, hyperresonance, asymmetric chest movement, hypoxemia, and signs of hemodynamic compromise.

Board Exam Tips for Recognizing a Pneumothorax

For respiratory therapy board exams, pneumothorax questions often focus on recognition and immediate management. The exam may describe a patient with sudden chest pain, dyspnea, unilateral decreased breath sounds, hyperresonance, and tracheal shift. This pattern should immediately suggest pneumothorax, especially tension pneumothorax.

In a stable patient, the best next step may be a chest x-ray to confirm the diagnosis. In an unstable patient with suspected tension pneumothorax, the correct action is immediate decompression. Do not choose imaging first when the patient is rapidly deteriorating.

For ventilator questions, look for sudden increases in peak and plateau pressures during volume-control ventilation. If the patient is on pressure-control ventilation, look for a sudden decrease in delivered tidal volume. Worsening oxygenation, hypotension, and unilateral absent breath sounds strengthen the suspicion.

Note: Remember that subcutaneous emphysema, crepitus, and air leaks in a chest tube drainage system may be associated with pneumothorax or ongoing pleural air leakage.

Pneumothorax Signs and Symptoms Practice Questions

1. What is a pneumothorax?
A pneumothorax is the presence of air in the pleural space, which can cause partial or complete collapse of the affected lung.

2. Why does a pneumothorax cause the lung to collapse?
Air entering the pleural space disrupts the normal negative pressure that keeps the lung expanded, allowing the lung to collapse.

3. What is one of the most common symptoms of a pneumothorax?
Sudden shortness of breath, or dyspnea, is one of the most common symptoms of a pneumothorax.

4. What type of chest pain is commonly associated with pneumothorax?
Pneumothorax often causes sudden, sharp, stabbing, or pleuritic chest pain on the affected side.

5. Why can a small pneumothorax cause severe dyspnea in some patients?
Patients with reduced pulmonary reserve, such as those with COPD or other lung diseases, may not tolerate even a small loss of lung function.

6. What breath sound finding suggests pneumothorax?
Diminished or absent breath sounds on the affected side suggest pneumothorax.

7. What percussion finding is commonly associated with pneumothorax?
Hyperresonance to percussion over the affected side is commonly associated with pneumothorax.

8. Why does pneumothorax produce hyperresonance?
Pneumothorax produces hyperresonance because excess air is present in the pleural space.

9. How does chest movement change with pneumothorax?
The affected side of the chest may move less than the unaffected side during inspiration.

10. What does asymmetric chest movement suggest in a patient with dyspnea?
Asymmetric chest movement may suggest pneumothorax, especially when paired with unilateral decreased breath sounds and hyperresonance.

11. What is subcutaneous emphysema?
Subcutaneous emphysema is air trapped in the soft tissues, often producing a crackling sensation called crepitus.

12. Does subcutaneous air always prove pneumothorax is present?
No. Subcutaneous air does not always prove pneumothorax, but it indicates that risk factors for an air leak are present.

13. What is tension pneumothorax?
Tension pneumothorax occurs when air becomes trapped under pressure in the pleural space and cannot escape.

14. Why is tension pneumothorax more dangerous than a simple pneumothorax?
Tension pneumothorax can compress the lung, shift the mediastinum, reduce venous return, lower cardiac output, and cause shock.

15. In tension pneumothorax, which direction does the trachea usually shift?
The trachea usually shifts away from the affected side.

16. What cardiovascular signs may occur with tension pneumothorax?
Tachycardia and hypotension may occur as venous return and cardiac output decrease.

17. Why does hypotension occur in tension pneumothorax?
Rising intrathoracic pressure reduces venous return to the heart, which lowers cardiac output and blood pressure.

18. What are key bedside signs of tension pneumothorax?
Key signs include diminished breath sounds, hyperresonance, tachycardia, and hypotension.

19. What additional trauma-related sign may be present in tension pneumothorax?
Jugular venous distention may be present due to impaired venous return from increased intrathoracic pressure.

20. Why should treatment not be delayed when tension pneumothorax is strongly suspected?
Tension pneumothorax can rapidly progress to cardiovascular collapse, so immediate decompression is required.

21. What ventilator change may suggest pneumothorax in a mechanically ventilated patient?
A sudden increase in peak airway pressure may suggest pneumothorax.

22. In volume-control ventilation, what pressure change may occur with tension pneumothorax?
Both peak and plateau pressures may suddenly increase due to decreased lung compliance.

23. In pressure-control ventilation, what change may suggest tension pneumothorax?
The delivered tidal volume may suddenly decrease because the lung is less compliant.

24. What oxygenation change may warn that a pneumothorax is worsening?
A falling oxygen saturation may indicate that the pneumothorax is enlarging or impairing gas exchange.

25. What should be done if a patient being observed for pneumothorax develops worsening respiratory distress?
The patient should be reassessed immediately, and drainage may be required if respiratory function is deteriorating.

26. What chest x-ray finding commonly indicates a pneumothorax?
A visible pleural line with no lung markings beyond it commonly indicates a pneumothorax.

27. Why are lung markings absent beyond the pleural line in pneumothorax?
Lung markings are absent because the space beyond the pleural line contains free air rather than expanded lung tissue.

28. What does a pneumothorax look like on a chest x-ray?
A pneumothorax often appears as a dark, radiolucent area around the collapsed lung with no visible lung markings.

29. What is the deep sulcus sign?
The deep sulcus sign is an abnormally deep and dark costophrenic angle that may suggest pneumothorax in a supine patient.

30. Why can pneumothorax be harder to detect on a supine chest x-ray?
In a supine patient, free pleural air may spread anteriorly and basally instead of rising to the lung apex, making the finding more subtle.

31. What imaging test is most sensitive for detecting pneumothorax?
A CT scan is the most sensitive imaging test for detecting pneumothorax.

32. When is CT especially useful in suspected pneumothorax?
CT is useful when the diagnosis is uncertain, especially in trauma patients, postoperative patients, or patients with bullous emphysema.

33. Why can bullous emphysema make pneumothorax harder to diagnose?
Large bullae can resemble pneumothorax on chest x-ray, so CT may be needed to distinguish between the two.

34. What ultrasound sign suggests pneumothorax?
Absence of lung sliding on ultrasound may suggest pneumothorax.

35. What is lung sliding?
Lung sliding is the normal movement of the visceral and parietal pleura against each other during breathing.

36. Why is lung sliding absent in pneumothorax?
Lung sliding is absent because air in the pleural space separates the pleural layers.

37. What is the lung point on ultrasound?
The lung point is the transition area where normal lung sliding meets the region of pneumothorax.

38. Why is the lung point important?
The lung point is important because it is highly specific for pneumothorax when identified.

39. What happens to B-lines in pneumothorax?
B-lines are typically absent in the area of pneumothorax.

40. Why are B-lines absent in pneumothorax?
B-lines are absent because pleural air separates the lung from the chest wall and prevents the artifacts from forming normally.

41. In a stable patient with suspected pneumothorax, what diagnostic test is commonly recommended?
A chest x-ray is commonly recommended to confirm the diagnosis in a stable patient.

42. In an unstable patient with suspected tension pneumothorax, should imaging be obtained first?
No. If tension pneumothorax is strongly suspected in an unstable patient, immediate treatment should occur before imaging.

43. What is the emergency treatment for tension pneumothorax?
The emergency treatment is immediate needle decompression, followed by chest tube placement.

44. What size needle is commonly used for adult needle decompression?
A large-bore needle, commonly 14 to 16 gauge, is used for adult needle decompression.

45. Where is needle decompression traditionally performed for tension pneumothorax?
It is traditionally performed at the second intercostal space at the midclavicular line on the affected side.

46. Why is the needle inserted over the top of the rib during decompression?
The needle is inserted over the top of the rib to avoid the blood vessels and nerves that run along the underside of the rib.

47. What does needle decompression do to a tension pneumothorax?
Needle decompression releases trapped pleural air and converts a tension pneumothorax into a simple pneumothorax.

48. Why is a chest tube usually needed after needle decompression?
A chest tube is needed to provide ongoing evacuation of pleural air and allow the lung to re-expand.

49. Where is a chest tube commonly inserted for pneumothorax?
A chest tube is commonly inserted into the fourth or fifth intercostal space at the midaxillary or anterior axillary line.

50. What does bubbling in the water-seal chamber suggest when a chest tube is treating pneumothorax?
Bubbling may suggest an active air leak from the pleural space.

51. What does it suggest when bubbling stops in the water-seal chamber after treatment for pneumothorax?
When bubbling stops, it may suggest that the air leak has sealed and the tissue tear has healed.

52. What is a sucking chest wound?
A sucking chest wound is an open chest injury that allows air to enter the pleural space through the chest wall.

53. Why can a sucking chest wound lead to pneumothorax?
It can allow outside air to enter the pleural space, disrupting negative pleural pressure and causing lung collapse.

54. What initial dressing may be used for a sucking chest wound?
An occlusive dressing, such as petroleum gauze, may be applied initially to help prevent further air entry.

55. Why is positive-pressure ventilation risky in an untreated pneumothorax?
Positive-pressure ventilation can force more air into the pleural space and worsen lung collapse or tension physiology.

56. What is the only absolute contraindication to IPPB therapy mentioned in the provided information?
Untreated tension pneumothorax is the only absolute contraindication to intermittent positive-pressure breathing therapy.

57. What mechanical ventilation pressures increase the risk of pneumothorax?
High airway pressures, especially pressures greater than about 40 to 45 cm H₂O, can increase the risk of pneumothorax.

58. Why are patients with emphysema at risk for pneumothorax?
Patients with emphysema may have blebs or bullae that can rupture and allow air to leak into the pleural space.

59. What is barotrauma?
Barotrauma is lung injury caused by excessive pressure, which can lead to air leaks such as pneumothorax.

60. What is volutrauma?
Volutrauma is lung injury caused by excessive volume delivery, which can contribute to alveolar rupture and pneumothorax.

61. What invasive procedure can accidentally cause pneumothorax?
Central venous catheter insertion through the subclavian or jugular vein can accidentally puncture the pleural space and cause pneumothorax.

62. Why is pneumothorax a concern after subclavian line placement?
The pleura may be accidentally punctured during insertion, allowing air to collect in the pleural space.

63. What should be suspected if a ventilated patient deteriorates after central line insertion?
Pneumothorax or tension pneumothorax should be suspected, especially if breath sounds decrease on one side and oxygenation worsens.

64. What does decreased tactile fremitus suggest in pneumothorax?
It suggests that air in the pleural space is blocking vibration transmission from the lung to the chest wall.

65. How does tactile fremitus in pneumothorax differ from consolidation?
Pneumothorax usually decreases tactile fremitus, while consolidation often increases tactile fremitus.

66. What does unilateral hyperresonance suggest on physical exam?
Unilateral hyperresonance strongly suggests excess air on one side, such as pneumothorax.

67. How can pneumothorax affect the position of heart sounds?
Heart sounds may shift away from the side of the pneumothorax, especially when mediastinal shift occurs.

68. Why are heart sounds shifting away important in neonates?
In neonates, breath sounds transmit widely through the small chest, so shifted heart sounds may provide a clue to pneumothorax.

69. Why is pneumothorax harder to recognize in neonates?
It is harder to recognize because breath sounds transmit widely through the small chest, making unilateral findings less obvious.

70. What bedside technique may help detect neonatal pneumothorax?
Transillumination with a high-intensity light may help detect pneumothorax in some neonates.

71. What does cyanosis suggest in a patient with pneumothorax?
Cyanosis suggests severe hypoxemia and inadequate oxygenation.

72. What does pulsus paradoxus suggest in tension pneumothorax?
Pulsus paradoxus may occur when increased intrathoracic pressure significantly affects venous return and cardiac output.

73. What rhythm may occur if severe tension pneumothorax is untreated?
Untreated severe tension pneumothorax can progress to pulseless electrical activity.

74. What does a rapid decline in cardiopulmonary status suggest after chest trauma?
A rapid decline after chest trauma may suggest tension pneumothorax, especially with unilateral breath sound changes and hypotension.

75. Why is tension pneumothorax considered a clinical diagnosis?
It is considered a clinical diagnosis because treatment may need to begin immediately based on bedside signs before imaging can be obtained.

76. What is the main difference between a simple pneumothorax and a tension pneumothorax?
A simple pneumothorax involves air in the pleural space, while a tension pneumothorax involves trapped air under pressure that compromises ventilation and circulation.

77. Why does mediastinal shift occur in tension pneumothorax?
Mediastinal shift occurs because rising pleural pressure pushes the heart, trachea, and other central chest structures away from the affected side.

78. What happens to venous return during tension pneumothorax?
Venous return decreases because increased intrathoracic pressure compresses the great vessels and limits blood flow back to the heart.

79. How does tension pneumothorax affect cardiac output?
Cardiac output falls because decreased venous return reduces the amount of blood available for the heart to pump.

80. Why can hypoxemia occur during pneumothorax?
Hypoxemia can occur because the collapsed lung is poorly ventilated, leading to impaired gas exchange and shunting.

81. What does worsening SpO₂ suggest during observation of a pneumothorax?
Worsening SpO₂ suggests deteriorating respiratory function and may indicate that the pneumothorax is enlarging.

82. What should be recorded when observing a patient with a known pneumothorax?
Respiratory rate, dyspnea, oxygen saturation, and signs of worsening respiratory function should be recorded.

83. What finding helps distinguish pneumothorax from pleural effusion during percussion?
Pneumothorax typically causes hyperresonance, while pleural effusion usually causes dullness to percussion.

84. What finding helps distinguish pneumothorax from atelectasis regarding tracheal shift?
Tension pneumothorax usually shifts the trachea away from the affected side, while severe atelectasis may pull it toward the affected side.

85. What should be considered when unilateral breath sounds are decreased in an intubated patient?
Pneumothorax, right mainstem intubation, mucus plugging, or unilateral lung disease should be considered.

86. What ventilator alarm may occur with pneumothorax?
A high-pressure alarm may occur due to decreased lung compliance and increased airway pressure.

87. Why can plateau pressure increase during tension pneumothorax?
Plateau pressure can increase because trapped pleural air compresses the lung and reduces lung compliance.

88. What does a sudden drop in delivered tidal volume suggest during pressure-control ventilation?
A sudden drop in delivered tidal volume may suggest decreased lung compliance from pneumothorax or tension pneumothorax.

89. Why is pneumothorax included in the differential diagnosis of sudden high airway pressure?
Pneumothorax can suddenly decrease lung compliance, causing airway pressures to rise during mechanical ventilation.

90. What chest radiograph position is preferred when pneumothorax is suspected?
An upright chest x-ray is preferred when possible because pleural air rises and is easier to identify.

91. What does increased radiolucency on chest x-ray mean in pneumothorax?
Increased radiolucency means the affected area appears darker because air is present where lung tissue markings should be seen.

92. Why is proper chest x-ray exposure important when evaluating pneumothorax?
Poor exposure can make the lung fields appear too dark or unclear, making pneumothorax harder to detect.

93. What does compression of the opposite lung suggest in tension pneumothorax?
Compression of the opposite lung suggests severe mediastinal shift from rising pressure on the affected side.

94. What can a depressed hemidiaphragm indicate in tension pneumothorax?
A depressed hemidiaphragm may indicate increased pressure and hyperexpansion on the affected side.

95. What patient population may experience dyspnea out of proportion to pneumothorax size?
Patients with reduced pulmonary reserve, such as those with chronic lung disease, may have severe dyspnea even with a small pneumothorax.

96. What is the affected lung expected to do when air enters the pleural space?
The affected lung collapses partially or completely toward the hilum.

97. Why should a chest radiograph be recommended in a stable patient with suspected pneumothorax?
A chest radiograph can confirm the presence of pleural air and help determine the size and severity of the pneumothorax.

98. What should the respiratory therapist do when clear signs of tension pneumothorax are present?
The respiratory therapist should recognize the emergency and support immediate decompression rather than delaying for imaging.

99. What signs should make a clinician suspect pneumothorax after blunt chest trauma?
Sudden dyspnea, inspiratory chest pain, unilateral decreased breath sounds, hyperresonance, and asymmetric chest movement should raise suspicion.

100. What is the most important exam takeaway about unstable tension pneumothorax?
If the patient is unstable and tension pneumothorax is suspected, treat immediately with decompression before obtaining imaging.

Final Thoughts

The signs of a pneumothorax can vary from mild shortness of breath to severe cardiovascular collapse. The most important findings include sudden chest pain, dyspnea, decreased or absent breath sounds on one side, hyperresonance to percussion, asymmetric chest movement, decreased tactile fremitus, falling oxygen saturation, tachycardia, and subcutaneous emphysema.

Tension pneumothorax adds more serious signs, including hypotension, tracheal deviation, jugular venous distention, worsening hypoxemia, and shock.

In stable patients, imaging can help confirm the diagnosis. In unstable patients with suspected tension pneumothorax, immediate treatment should not be delayed.

John Landry, RRT Author

Written by:

John Landry, BS, RRT

John Landry is a registered respiratory therapist from Memphis, TN, and has a bachelor's degree in kinesiology. He enjoys using evidence-based research to help others breathe easier and live a healthier life.